Publications
Beinfeld MC, Blum A, Vishnuvardhan D, Fanous S, Marchand JE. Cholecysokinin (CCK) levels in prohormone convertase 2 knockout mouse brain regions reveal a complex phenotype of region-specific alterations. J Biol Chem 2005 Nov 18;280(46):38410-5.
Pratt JS, Blum A, Vishnuvardhan D, Kitagawa K, Beinfeld MC. Cleavage-site mutagenesis alters post-translation processing of Pro-CCK in AtT-20 cells. Biochemistry. 2004 Jul 27;43929):9502-11.
Cain BM, Connolly K, Blum AC, Vishnuvardhan D, Marchand JE, Zhu X, Seiner DF, Beinfeld MC. Genetic inactivation of prohormone convertase (PC1) causes a reduction in cholecystokinin (CCK) levels n he hippocampus, amygdala, pons, and medulla in mouse brain that correlates with the degree of colocatization of PC1 and CCK mRNA in these structures in rat brain. J Neurochem. 2004 apr;89(2):307-13.
Cain BM, Connolly K, Blum A, Vishnuvardhan D, Marchand JE, Beinfeld MC. Distribution and colocalization of cholecystokinin with the prohormone convertase enzymes PC1, PC2, and PC5 in rat brain. J Comp Neurol. 2003 Dec 15;467(3):307-25.
Beinfeld MC, Vishnuvardhan D, Blum A, Reynolds N, Fannous S, Kitagawa K, and Marchand JE. Inhibition of prohormone convertase 1 (PC1) expression in cholecystokinin (CCK) expressing At-T20 cells decreased cellular content and secretion of CCK and caused a shift in molecular forms of CCK secreted. Peptides 27:905-910.
Reynolds NA, Blum A, Kitagawa K and Beinfeld MC. Inhibition of PC5 expression decreases CCK secretion and increases PC2 expression. Peptides 27:901-904.
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